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  • Can Parkinson’s disease be prevented? : epidemiological evidence on lifestyle factors 

    Yang, Fei (Inst för medicinsk epidemiologi och biostatistik / Dept of Medical Epidemiology and Biostatistics, 2017-03-24)
    The etiology of Parkinson’s disease (PD) is largely unknown, but epidemiological studies have provided robust evidence that environmental factors have a central role in PD pathogenesis and progression. The studies included ...
  • Identifying individual antibiotic treatments prescribed for mothers and for children up to the age of five following specific inpatient diagnoses 

    Almqvist, C.; Örtqvist, A.K.; Gong, T.; Wallas, A.; Ahlén, K.M.; Ye, W.; Lundholm, C. (Inst för medicinsk epidemiologi och biostatistik / Dept of Medical Epidemiology and Biostatistics, 2017-03-20)
    Aim: Exposure to antibiotics in early life may affect future health. Most antibiotics are prescribed in outpatient care, but inpatient exposure is also important. We estimated how specific diagnoses in hospitals corresponded ...
  • Increased mortality in patients with congenital adrenal hyperplasia due to 21-hydroxylase deficiency 

    Falhammar, H.; Frisén, L.; Norrby, C.; Linden Hirschberg, A.; Almqvist, C.; Nordenskjöld, A.; Nordenström, A. (Inst för medicinsk epidemiologi och biostatistik / Dept of Medical Epidemiology and Biostatistics, 2017-03-20)
    Context: Reports on mortality in patients with congenital adrenal hyperplasia (CAH) are lacking. Objective: To study mortality and causes of death in CAH. Design, Setting and Participants: We studied patients ...
  • Similar familial risk in multiple sclerosis subgroups Introduction 

    Song, J.; Karrenbauer, V.; Manouchehrinia, A.; Almqvist, C.; Hillert, J.; Westerlind, H. (Inst för medicinsk epidemiologi och biostatistik / Dept of Medical Epidemiology and Biostatistics, 2017-03-20)
  • Gut microbes and the developing brain 

    Arentsen, Tim B. F. (Inst för neurovetenskap / Dept of Neuroscience, 2017-03-17)
    The discovery that commensal gut microbiota can influence host development and physiology beyond the gastrointestinal (GI) tract has triggered a paradigm shift in our conceptualization of the origin of human diseases. A ...

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